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Rich,

Oxygen is a vasoconstrictor, at 2.0 ATA we are talking about a 17% decrease
in inert gas elimination secondary to this reduced blood flow.

This obviously is not a major reduction or constrictor effect, but it 
is a vasoconstriction effect that is mediated by elevated partial pressures
of oxygen.

Some work has been done at Duke (I think it was by Dick Vann) on using
intravenous vasodilators to offset this effect (I think they were using 
isuprel IV, which has both a vasodilation effect and a marked positive 
inotropic effect, so we really do not know if the positive inotropism
increased blood flow to offset the oxygen vasoconstriction, or if the
vasodilation from the isuprel did the trick, or if it was a combination 
of the two effects.).

John 

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