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To whom is interested, We have established, thanks to A. Appleyard, that it is possible for the Superoxide anion to be forced off by other O2 molecules with a high ppO2 in the body. The reactions contolling this process are designed to not let this happen under normal conditions, but it is concievable for this to happen under high ppO2 conditions. Now, I have also found out that the Superoxide anion is normally produced in small concentrations during the oxidation of iron(Fe2+ to Fe+3) in heme (the O2 carrier for hemeoglobin). As mentioned before, again thanks to A. Appleyard,there are enzymes to rid the body of these destructive compounds. But we know if there is an overwhelming amount of anions to be then there will be an accumulation of them; therefore, possibly causing O2 toxicity. Now the question here is, does the root of this condition really happen from the electron transport chain or from the oxidation of iron for heme? Also are there any other reactions that could contribute to this condition? My guess is that the condition is an accumulation of multiple aspects which were described earlier and even other things not discussed! Aloha Tony ANTHONY MONTGOMERY Marine Option Program Waikiki Aquarium 1000 Pope Road MSB #203 2777 Kalakaua Ave Honolulu, Hawaii 96822 Honolulu, Hawaii 96815 Fax: (808) 956-2417 Fax: (808) 923-1771 Phone: (808) 956-6000 Phone: (808) 923-9741
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