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At 7:05 PM 9/19/95, Richard Pyle wrote:
>> I've checked with the archives, the litrature, my surgeon, friends who are
>> commercial divers and under the bed, but have not found an authoritative
>> study that confirms the popular opinion that injury sites are more prone to
>> DCS than uninjured body parts.
>
>It was explained to me by my "bends doc" that I was more susceptible to
>future DCI primarily because my previous hit left me with scar tissue,
>and scar tissue has poor circulation, making it an *extremely* "slow"
>tissue. Other than that, I know of no other references to correlating
>scar tissue or other sites of healed damage to increased susceptibility
>to bubble formation/trapping.  I'd be interested to know what you find
>out more on this topic.

This is exactly the same story I've heard, but when I talked to my surgeon
about it, he smiled and sayed, 'so if the circulation is poor, what effect
will that have on the ongassing rate?'  I started thinking about it and could
see his point; all things being equal, both ongassing and offgassing rates
will be effected by poorer circulation.  I guess the question then becomes,
is scar tissue a 'new' compartment, or are their other tissues just as slow
or slower?

So the stories persist, and my surgeon wasn't a dive doc (he's a knee
man).

Scott.

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