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> 3) the vaso constricting effect of
> > the high ppo2 is offset enough to open up the
> > vessels in the tissues ( and the capillary beds of
> > the lungs) for more rapid offgassing.
>
>So here's my question. I have yet to find an instance where something
>that Trey said was (verifiably) wrong.
Neither have I, that is one of things that brought me to the WKPP.
> That's not a shot, that's
>sincere. So likely I'm misunderstanding something here. It sounds like
>Trey is saying that the breaks are increasing efficiency by reversing
>vasoconstriction, yet you say the breaks aren't long enough for that to
>happen plus the vasoconstriction does not really impair the offgassing
>at the PPO2's that we're talking about (1.6 and less). So can either
>you or Trey clear up my confusion?
Reread the statement he made a little more carefully. He said it "is
offset enough to open up the vessels" This should not be enough to
completely "reverse" (return to complete normalcy) the constriction per se,
that might form at normal levels of deco i.e. that is outside of multiple
hours at 300ft , because it is not severe. I am sure there is a mild
constricting effect at normal diving PO2s, but this is sufficiently offset
by the ability of the oxygen to continue to diffuse in spite of the
constriction, and should also be relieved (not reveresed) by breaks.
Also keep in mind what I said in the first post is geared toward diving
that does not have the massive exposures found in the WKPP. With the
longer exposure times the possibility is higher and the benefits derived,
from breaks, etc., are more pronounced.
Did this provide some clarification?
Scott
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