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On Fri, 23 Dec 1994, David Doolette wrote: > Compression on air, as in > Richard's anecdotes, will have two beneficial effects. Firstly, it will > compress any bubbles, although, as I have posted numerous times, this effect > will be small V=4/3 (pi r^3). This brings up a question I've always had...does anyone have any insight on whether it is the diameter, surface area, or volume of a bubble that has the most impact on symptom severity? I know there is no easy answer, and I suspect all three parameters have an effect (difficult to tease appart with case studies & experiments, but it might seem surface area should have the greatest impact with regard to complement effects). > between tissue bubbles and alveolar/venous gas. With increasing gas phase > separation, the oxygen window tends to drive off-gassing and bubble > shrinkage. For more information, buy my book. Wait now... YOU have a book too? What's the title, how much, and can I send a single bank draft for both yours & Rat's? > Thus, the hydrostatic pressure does matter. Compression of bubbles may > reduce mechanical distortion of tissues and rheological changes. > Compression even on air will be better than surface air breathing for bubble > resolution, but not better than NBO or HBO which provides a bigger oxygen > window. By "NBO" (new to me), I assume you mean breathing O2 on surface? I recognize the theoretical advantage of surface O2 over air under pressure. My question to Rat (and anyone else who wants to chime in), was whether all the apparent success cases of air-only IWR would have been better-off breathing O2 on the surface? Theory says yes (oxygen window), but I'm not so sure... Again, this is NOT a pointed question. It is also a rather rhetorical one, since, of course, nobody can say whether these people would have been better-off one way or the other. I'm looking forward to receiving my copy of the current "Alert Diver" issue, to see how DAN suggests a controlled study would need to be structured. Aloha, Rich deepreef@bi*.bi*.ha*.or*
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