A quick follow up regarding some additional information gleamed from a quick search of other studies investigating the occurrence of PFO and decompression sickness, especially given George's "gentle" comdemnation of my last post suggesting PFO may not be a factor associated with decompression sickness. Before I go further for the sake of the list I retract any suggestion I may have made regarding PFO not being associated with an increased risk of decopression sickness. In my haste of seeing an article relevant to a thread on the list I posted it without doing alittle background check to ensure its validaty. Thanks to George for his comments and provoking me to look beyond that single article. I only post this stuff so people don't only remember the one study suggesting PFO does not increase your chances of suffering decompression sickness. I won't waste your time and bandwidth with my own opinions on the studies listed below, as any reasonable person should be able to realise what they mean and what each individual diver should be doing to reduce their risks. If you're uncertain then reread George's rebuttal to my initial post. Bove (1998) found using statistical analysis of subjects from sport diving, US navy and comercial diving populations that the presence of a PFO produces a 2.5 times increase in the odds for developing serious (type II) DCS in all three types of divers. Unfortunately the author then suggests that as the odds ratio for developing DCS is small anyway, the risk of devloping DCS in the presence of a PFO remains small and therefore doesn't warrant routinue screening by echocardiography of sport, military or commercial divers. Before you hit that reply button read on... Germonpre et al (1998) compared the presence of a PFO in belgian divers that had suffered from neurological DCS with a control population that had not suffered any DCS. The statistics are staggering: Of divers suffering from neurological DCS 59.5% had a PFO while the only 36% of control divers had a PFO. For divers suffering cerebral DCS 80% had a PFO compared to 25% in controls and for divers with spinal DCS there was no differences between those with DCS and the control populations. When divers with UNEXPLAINED cases of DCS were examined 83% had a grade 2 PFO (grade 2= score of greater or equal to 20 bubbles contrast passage at rest or after Valsalva strain). To summarize, this study suggests that a PFO is strongly correlated with cerebral DCS. The authors also noted that there was a striking difference in the method used for middle ear equalization between divers suffereing cerebral and spinal DCS. Whereas spinal DCS divers had no problems equalizing (yawning or light Valsalva), the divers suffering from cerebral DCS reported they had to "push hard" to clear their ears. This might be relevant as the Valsalva maneuver induces a major rise in right atrial pressure, a major cause of blood shunting thru the PFO. They recomend divers with unexplained DCS or with cerebral DCS hits be investigated for PFO. If a grade 2 PFO is present then those divers are advised to follow dive profiles that are very low "bubble-prone" (perhaps the list might want to comment on what sort of dive this might be???? ie. <3m) or give up diving. I won't bore you all to tears with details of the results of animal models except to say that using anesthetized pigs with PFO the risks of arterial bubbles and air embolism is higher. Phil Davies Bove AA (1998) Risk of decompression sickness with patent foramen ovale. Undersea Hyperbar Med 25:175-178. Germonpre, P Dendale, P unger P and Balestra C (1998) Patent foramen ovale and decompression sickness in sports divers. J. Appl Physiol 84:1622-1666. -- Send mail for the `techdiver' mailing list to `techdiver@aquanaut.com'. Send subscribe/unsubscribe requests to `techdiver-request@aquanaut.com'.
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