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To: Richard
To: Pyle <deepreef@bi*.bi*.ha*.or*>
To: techdiver@opal.com
Subject: Re[3]: Australian O2 protcol.
From: <bob.talbot@ae*.or*.uk*>
Date: Thu, 15 Dec 1994 10:25:57 +0000
#He thinks the rhythmic compressions from passing swells somehow increased 
#inert gas elimination or decreased bebble growth/formation.  I'd like to 
#see some controlled studies on that one, but Phil's anecdotal experience 
#rates almost equally with controlled scientific study in my book.

Simplistically, bubbles in the blood are no problem as long as they keep moving.
Only when arterial bubbles get lodged, cutting off the supply to somewhere vital
(like portions of the brain?) do they start to become clinically significant.

Just maybe, were the anecdotal observation a true record, the rhythmic 
compressions serve to dislodge rather than necessarily remove those trapped 
bubbles from where they were causing a problem?

Of course, I dont see how this could affect sympoms due to autochthonous 
(tissue) bubbles .... 

Any experimental evidence available on rhythmic compressions ... such as during 
reco. of bent rabbits, goats, pigs etc?

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