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Kevin,

The problem is that the human body is designed to work with a arterial
CO2 of approx. 40torr.  Yes, we can drive the arterial CO2 levels down
to the point that a patient will not spontaneously breathe, but we are
changing some very delicate ion balances when we do this (lets not
get into the Henderson-Hasselbach equation, buffers, bicard, the kidneys,
etc.).

Bottom line is, if we maintain arterial pCO2 at a normal level (ie, 40torr),
we will get breathing efforts in the vast majority of normal patients.

John

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