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Rich, no need for you to be on the bandwagon, you don't dive air anyway, so believe what you please.My Chinese reference was to bubbel mehcanics, the same thing you like to use - the Chieese mathematicians have a fantastic understanding of this, but this is not the subject. However, a note of correction here: the damage by the passage of the rigid rbs'c sets up a sequence of events including triggering stretch receptors in the vessels, releasing chemicals like nitric oxide, and other reactions that set up an immune response ( stuck bubbles would do the same thing, Richie, so stay off the bandwagon) that reustls in you ffeeling the "flulike symptoms". We all know that this occurs with air more easily, and we all now know to do deep sotps with all gases, but then we do not dive deep air anymore anyway. Those who do get the "flu". Ask the Navy, they spotted it first. Good to hear from you , Richie, now shut up:). Richard Pyle wrote: > > Bill, > > Excellent post! Couple questions: > > > Since the capillary micro-circulation plays an essential role in gas > > transfer during compression and decompression one might be further > > concerned as to the effects of red blood cell rigidity associated with > > hyperbaric exposures to air. > > Wouldn't you think the biochemical consequences of the complement system > response would likely have a more direct impact on manifestation of DCI > (or other "flulike") symptoms, than simple reduced perfusion leading to > impaired gas transport at the critical tissue sites? > > Also, do you have any idea where I might find the primary literature > concerning this whole notion of RBC rigidity in response to high PN2? > I've been trying to get the answer from George ever since he started > talking about it, but the best I can get out of him is some cryptic > reference to "Chinese researchers". I don't doubt the effect - I just > would like to find where and how it was elucidated. > > > There was some speculation recently in the DAN journal > > regarding "Divers Disease", a flulike illness which sometimes > > accompanies hypberbaric exposures. I wouldnt be at all surprised if the > > cause of this problem turns out to be the aftereffects of red blood > > cell rigidity and the consequent build up of haemolized cells in the > > spleen and lymph nodes. > > I have experienced these sorts of symptoms following deep air dives many, > many times in the past (along with excessive fatigue). However, they > disappeared entirely when I began doing slower deep ascents (including > those from "deep" air dives). It seems to me that this phenomenon might > be more a function of bubble growth and consequent compelment effects, > rather than a result of haemolysis due to RBC rgidity during the high-PN2 > portion of the dives. > > > For the mean time I think its a safe bet to avoid deep air at all costs. > > I wholeheartedly agree, but more because of the real cause of diving > accidents (pilot error, perhaps exacerbated by narcosis), than the > potential consequences of RBC rigidity, if it exists. > > Aloha, > Rich > > P.S. I want to emphasize that I don't necessarily doubt the RBC rigidity > thing due to elevated PN2; I just want to find the source before I jump > on the bandwagon. > > Richard Pyle > Ichthyology, Bishop Museum deepreef@bi*.bi*.ha*.or* > 1525 Bernice St. PH: (808) 848-4115 > Honolulu, HI 96817-0916 FAX: (808) 841-8968 > "The views are those of the sender and not of Bishop Museum" > > -- > Send mail for the `techdiver' mailing list to `techdiver@aquanaut.com'. > Send subscribe/unsubscribe requests to `techdiver-request@aquanaut.com'. -- Send mail for the `techdiver' mailing list to `techdiver@aquanaut.com'. Send subscribe/unsubscribe requests to `techdiver-request@aquanaut.com'.
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