Can anyone shed light on this corn ball theory of mine? Having read Richard Vann's article on O2 exposure management in Aquacorps I think I understand that if O2 enters the respiratory chain prematurely molecules such as superoxide anion and hydrogen peroxide can form, if the O2 is at excess pressure these can overwhelm the deactivating superoxide dismutase and catalase. This is part of the O2 problem. Now . . . .here it comes, are superoxide anions and hydrogen peroxide what we know as free radicals? and if they are can this effect be countered partially by the intake of quantities of anti-oxidants as are found at drug stores? or is this all hokum? Thanks Andrew Poole DiveTech "Live long and Scuba"
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