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Date: Mon, 12 Jan 1998 14:13:22 -1000 (HST)
From: Richard Pyle <deepreef@bi*.bi*.ha*.or*>
To: "William M. Smithers" <will@tr*.co*>
Cc: Ben Greenhouse <b.greenhouse@ut*.ca*>,
     "techdiver@aquanaut.com" ,
     rebreather@nw*.co*
Subject: Re: Surface O2 and DCS...
> There's still direct dissolution in the blood and tissues
> at elevated PO2, just like there's direct dissolution of N2
> at elevated PN2's, though unlike N2 or He, as Rich points out, 
> it's probably reduced somewhat by direct metabolic consumption.

I'm not sure if the unbound dissolved O2 is consumed directly, or if 
metabolic use of Hb-bound O2 frees up more Hb binding sites, to which the 
unbound O2 may bind. There are complicating factors (e.g., Bohr effect), 
but the point is that O2 is being removed (in one way or another) by 
metabolism; whereas He and N2 are not.

> Hence, presumably because of the benefits of direct tissue 
> consumption of O2, we see the rising popularity of 
> alternative medical uses (besides unbending divers), of high 
> PO2 therapy. 

.....until they discover the descructive effects of oxidation, which might 
explain aging.....

Aloha,
Rich
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