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On Thu, 8 Sep 1994, J Shepherd wrote: > Rich calls this STB, I've called it sub clinical DCS, Rich feels > that it's slow tissues (classic bends) I and at least one other person > feel that it's more likely to be something else (sorry I've forgotten > who it was, but they were talking about type II [neurological?] > symptoms). All the ingredients for a classic debate. > The only reason we call it "slow tissue blues" is that it seems to be most associated with profiles that lead to a long N2 soak (on an EDGE computer, the limiting compartments on such dives are always more towards the right of the screen - the slow side). I have no idea whether there is any physiological basis to this, and my evidence is very esoteric, so I'll gladly take a back seat to people with a clearer understyanding of diving physics/physiology as to what's really going on. (sorry - you won't get much debate out of me on this one... ;-> ) > Here's my thought; several DCS planning systems use a 'slab' > concept, that being that rather than several seperate tissues, you treat > the body as a slab of tissue with the fastest and most resilient tissues > exposed to the air, and the slowest and most susceptible (critical > ratios lowest) furthest away. > Could we be seeing the release over time of N2 from the slower > parts of the 'slab' into the faster parts, maybe as dissolved N2, maybe > as microbubbles? That would explain the time the symptoms take to pass > (several hours), yet sits easier with me wrt the site of action e.g. > blood or brain. That sounds good to me....consistent with our experiences. Another interesting topic.... Aloha, Rich deepreef@bi*.bi*.ha*.or*
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