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	Just to sling a little more juice into this fascinating tale;

	Rich calls this STB, I've called it sub clinical DCS, Rich feels
that it's slow tissues (classic bends) I and at least one other person
feel that it's more likely to be something else (sorry I've forgotten
who it was, but they were talking about type II [neurological?]
symptoms). All the ingredients for a classic debate.

	Here's my thought; several DCS planning systems use a 'slab'
concept, that being that rather than several seperate tissues, you treat
the body as a slab of tissue with the fastest and most resilient tissues
exposed to the air, and the slowest and most susceptible (critical
ratios lowest) furthest away. 

	Could we be seeing the release over time of N2 from the slower
parts of the 'slab' into the faster parts, maybe as dissolved N2, maybe
as microbubbles? That would explain the time the symptoms take to pass
(several hours), yet sits easier with me wrt the site of action e.g.
blood or brain.

	The comment about sleep responses to flu sounds convincing too.

	Jason.

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