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Date: Mon, 03 Nov 1997 18:17:49 -0500
From: "G. Irvine" <gmirvine@sa*.ne*>
Organization: Woodville Karst Plain Project
To: meademac@nh*.ul*.co*
CC: cavers <cavers@ge*.co*>, Ben Greenhouse <b.greenhouse@ut*.ca*>,
     techdiver@aquanaut.com
Subject: Re: Physiology
This is why we do not dive air deep, and would explain the spinal and
brain lesions, as well as the subclinical DCS symptoms following air
dives beyond the usual zone. On the other hand , we don't experimant
with powrful drugs under pressure, especially when conditioning and
non-narcotic mixes will produce the same result. I realize that this is
out of the question for some of the more vocal deep air promoters, but
then this fits perfectly : it takes a lot of work to do real tech
diving, and all it takes to deep air dive is stupidity and a place to
jump in.


Meade McCrory wrote:
> 
> Ben and others,
> 
>   I think a more relevant discussion of high ppN2 and RBC rigidity
> should include tissue oxygenation. The bi-concave shape of RBC's is
> designed to allow the RBC to twist and deform to enter areas of the
> microcirculation.There are pharmacologic means to enhance this ability
> of the RBC's to perform this function eg.)pentoxifylline-to improve
> oxygenation in a diagnosis of intermittent claudication.
>   In my opinion this seems to be more important than it's possible
> hypertensive effects.Has anyone looked at this before?
> 
> Meade McCrory
> Pharmacist/Dive Inst/Blue Hole dreamer
> 
>
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