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> scuba <scuba@uc*.be*.ed*> wrote on Fri 5 Aug 1994 > (Subject: Bent fish, terrorist dolphins & human evolution...):- > > > ... Suunto dive computer on some of the untrained Miskito Indian lobster > divers ... typical profile ... 3 dives. 198 ft for 43 mins, [then] 100 ft for > 80 mins, [then] 103 ft for 20 mins [6 tanks used with no decomp stops].... > then ... lunch break [and similarly in the afternoon]. ... he should be dead > ... The resistance is believed to be due to a blood protein that protects them > from the immediate symptoms of DCS. ... Appleyard commented: > I read in a reader's letter to a UK sport diving magazine a theory that > "bends bubbles tend to form around the small blood cells called platelets, and > that during steady diving this process with `silent bubbles' keeps the blood > clean of platelets as fast as platelets form, but if he stops diving for a > while and the blood gets a platelet load he is more liable to serious bends in > his first few dives after resuming diving". How does this differ from 'micro-nuclei' being 'used up' by silent bubble formation during regular, intensive diving, giving short term 'immunity' to DCS? Is it the lack of platelets, hence reduced chances of bubbles causing coagulation or lack of bubble-forming micro nuclei which possibly reduces the risk of DCS? I find it difficult to imagine that this process is the only mechanism involved in reducing the M.Indians's DCS hits. It would be interesting to see if they actually 'bubble' less than us mere mortals. Maybe they are just more tolerant than us (mind over matter)? Philip ******************************************************************** Philip Coetzee * E-Mail: zlapsc@zo*.up*.ac*.za* Department of Zoology * Phone: +27-41-5042138 University of Port Elizabeth * Fax: +27-41-5042317 P O Box 1600 * Port Elizabeth 6000 * SOUTH AFRICA * ********************************************************************
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